Consistently的問題，透過圖書和論文來找解法和答案更準確安心。 我們找到下列懶人包和總整理

A Home for Unwed Husbands

為了解決Consistently的問題，作者 這樣論述：

WINNER of a Pushcart Prize, The Flannery O’ Connor Prize and The Leapfrog Global Fiction Prize.At forty-four, Kay Sorensen has quit drinking, smoking, and overeating, and she has almost quit reading self-help books about quitting drinking, smoking and overeating. She has divorced her deadbeat hus

band, finished college, and landed a job she loves directing a small branch of the county library. But Kay still has one unconquered addiction: she just can’t say no to someone who needs her. So when her architect father insists he needs her to move back home and care-take the empty house she grew u

p in, Kay is forced to return to the site of her bitterly unhappy childhood, trying her best to ignore the ghostly presence of her dead mother and make a home for her and her son.But she soon finds herself returning to the patterns of her childhood, where she spent years trying to please her thankle

ss father and placate her invalid mother. When her dogmatic, born-again brother arrives, along with a slew of men from her past all seeking a home, Kay is suddenly playing housewife and host to five men with needs and demands she is struggling to meet while consistently ignoring her own.In order to

find freedom and regain her selfhood, Kay must travel halfway across the world, finally face the chattering ghosts of her past, and break out of the mold that has been set for her by the men who have controlled her whole life.

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考慮非完全游離針對隨機參雜之電晶體之電流電壓 變異性分析

為了解決Consistently的問題，作者曾郁鈞 這樣論述：

根據摩爾定律的延續，電晶體在晶片裡的密度每 兩年即倍增，也因此提升工作時的表現和降低能量的消 耗。而電晶體運作時的電流機制是建立在假設電位和雜質濃度是連續的情況下的飄移 擴散模型。當電晶體隨著科技的進步發展至奈米等級的結構時，許多可靠度的問題 隨機參雜 會因此被放大，甚至破壞 原本漂移 擴散模型的假設。因此在探討這方面的問題前，我們必須要對隨機參雜的雜質做深入的探討，並且發展一個物理模型來解決 此 問題。然而，典型的物理模型卻只能考慮數量對電晶體造成的影響，而無法將雜質位置對電晶體的影響正確地考慮進去。除此之外，在典型的元件模擬中，雜質的游離率都 假設 為 100% 。但實際上在高雜質濃度

的條件下是不符合的。在高雜質濃度的條件下亦會產生能隙縮減的量子效應，進而影響了電晶體的表現。因此，為了要得到更準確的模擬結果，同時考慮這兩項因素是必須的(非完全游離&能隙縮減模型)。然而，此模型是一束縛態問題，而飄移-擴散模型是非束縛態的問題，因此不容易在典型的飄移擴散模型上考慮此模型。在此論文中，我們設計了一套新的方法，可以在飄移-擴散模型的前提下考慮隨機參雜(雜質數目、雜質位置)的影響，且同時計算出雜質的游離率和能隙縮減的量。接著利用蒙地卡羅方法探討在平面電晶體的電流電壓的變異性。

How Russia Gets Its Way: And What the West Can Do about It

為了解決Consistently的問題，作者Giles, Keir 這樣論述：

You may not be interested in Russia. But Russia is interested in you. Russia’s 2022 attack on Ukraine saw confrontation between Moscow and the West spill over into open conflict once again. But Russia has also been waging a clandestine war against the West for decades. Hostile acts abroad, from p

oisoning dissidents to shooting down airliners, interfering in elections, spying, hacking and murdering, have long seemed to be the Kremlin’s daily business. But what is it all for? Why does Russia consistently behave like this? And what does it achieve?In this book, Keir Giles explains how and why

Russia pushes for more power and influence wherever it can reach, far beyond Ukraine - and what it means not just for governments, but for ordinary people. Bringing together stories from the military, politics, diplomacy, espionage, cyber power, organised crime and more, Giles describes how Moscow c

onducts its campaigns across the globe, and how nobody is too unimportant to be caught up in them. By lifting the lid on the daily struggle going on behind the scenes to protect governments, businesses, societies and people from Russian hostile activity, Russia’s War On Everybody shows how Moscow’s

hostile intentions for the rest of the world are far broader and more ambitious, and the ways it tries to achieve them far more pervasive and damaging, than we realise.

透過基於基因本體之整合性分析識別卵巢上皮性腫瘤發病機轉的失調基因功能體

為了解決Consistently的問題，作者蘇國銘 這樣論述：

上皮性卵巢癌（EOCs）在晚期或復發的婦科惡性腫瘤中常是致命的和頑固的，其中漿液性佔絕大多數而卵巢清亮細胞癌（OCCC）是僅次於漿液性上皮性卵巢癌的第二常見的上皮性卵巢癌。即便經過腫瘤減積手術後加上化學藥物治療後仍有不少的患者有著較差的預後或是復發，故整體而言，對於卵巢癌的治療仍是一個相當大的挑戰。此外，邊緣性卵巢腫瘤(BOT)，包括漿液性 BOT與黏液性BOT，是屬於介於良性與惡性之間的卵巢疾病，雖然大部分的預後不差但是也有與卵巢癌不同的組織病理學特性。本研究使用以基因本體(GO)為基礎加上機器學習輔助運算的綜合分析去探討卵巢清亮細胞癌以及漿液性卵巢腫瘤包含漿液性邊緣性卵巢腫瘤與漿液性卵巢

癌的GEO資料庫中失調的基因體、功能途徑，藉以去識別重要的差異表達基因(DEG)。首先在卵巢清亮細胞癌的整合性分析中，發現無論是早期抑或是晚期，與免疫功能相關尤其是活化補體系統的替代途徑的功能失調在腫瘤發生佔有相當重要的關聯性，而補體C3與補體C5也影響了疾病無惡化存活期(Progression-free survival, PFS)和整體存活率(Overall survival, OS)且免疫染色結果是有意義的。而在漿液性卵巢腫瘤的分析中發現，SRC基因和功能失調的芳烴受體(AHR)結合路徑(Binding pathway)確實影響PFS和OS，而且與上皮細胞間質轉化(Epithelial-

mesenchymal transition, EMT)相關的鋅指蛋白SNAI2在腫瘤發生過程中有重要角色，並顯示出從漿液性 BOT 到卵巢癌有著逐漸上升的影響趨勢。未來，標靶治療可以專注於這些有意義的生物標誌並結合精確監測，以提高治療效果和患者存活率。