歡迎來到演算法、軟體和硬體三位一體的未來世界論文書籍站
Adenoid tonsil的問題，透過圖書和論文來找解法和答案更準確安心。 我們找到下列懶人包和總整理
Adenoid tonsil的問題，我們搜遍了碩博士論文和台灣出版的書籍，推薦寫的 Pediatric Otolaryngology for Primary Care 和Lehr, Teresa K.的 The Great Tonsil Massacre都 可以從中找到所需的評價。
這兩本書分別來自 和所出版 。
長庚大學 臨床醫學研究所 邱政洵所指導 黃建嘉的 兒童鼻咽部微生物及免疫病理學研究 （2019），提出Adenoid tonsil關鍵因素是什麼，來自於介白素17A、肺炎鏈球菌帶菌、睡眠呼吸障礙、中耳積液、腺樣體增生、微生物相。
而第二篇論文國立陽明大學 分子醫學博士學位學程 黃奇英所指導 呂學儒的 利用次世代基因定序找尋轉移復發頭頸癌之預測因子 （2019），提出因為有 next-generation sequencing、head and neck cancer、predictors、synthetic lethality的重點而找出了 Adenoid tonsil的解答。
Pediatric Otolaryngology for Primary Care
為了解決Adenoid tonsil 的問題，作者 這樣論述：
This popular AAP manual provides expert help for virtually any commonly encountered pediatric ENT condition.Pediatric Otolaryngology is the first place to turn for expert help with the myriad ear, nose, and throat disorders. From adenopathy to ankyloglossia, ear infection to epistaxis, and from stri
dor to speech disorders, look here for the state of the art across the length and breadth of pediatric ENT disorders. Pediatric Otolaryngology delivers the precise level of detail primary care providers require. The book's practice-focused, case-based presentation streamlines patient problem-solving
. "How to" clinical pearls help you master key diagnosis and treatment procedures. This must-have manual includes more than 100 high-quality photos and illustrations to aid with diagnosis.KEY FEATURESThree new chapters: Complications of Chronic Eustachian Tube Dysfunction: Retractions, Perforations,
Cholesteatoma; Feeding Disorders/Ankyloglossia; and Pediatric TracheostomyUpdated guidelines for treatment of otitis media with effusionUpdated guideline on newborn hearing assessment and cytomegalovirus causationUpdated recommendations to treat intractable drooling Scott R Schoem, MD, MBA, FAAP
is a graduate of Georgetown University School of Medicine. He performed his surgical internship and otolaryngology residency at the National Naval Medical Center in Bethesda, MD and his fellowship in pediatric otolaryngology at National Children’s Medical Center in Washington, DC. He founded and has
been Director of Otolaryngology at Connecticut Children’s Medical Center in Hartford, CT since 1999. Dr Schoem is a Professor of Clinical Otolaryngology at UCONN, the Past President of the CT ENT Society, and the Past Chair of the Section on Otolaryngology - Head and Neck Surgery of the American Ac
ademy of Pediatrics (AAP). He is the President-elect of the CT AAP state chapter. Dr Schoem is active in children’s healthcare advocacy and policy. He is a board member of ENT PAC at the American Academy of Otolaryngology - Head and Neck Surgery. He has extensive experience lobbying both at the stat
e and federal level to members of Congress. David Darrow, MD, DDS, FAAP is a Professor of Otolaryngology and Pediatrics at the Eastern Virginia Medical School, and an attending otolaryngologist at the Children’s Hospital of The King’s Daughters in Norfolk, VA. After receiving degrees in both dentist
ry and medicine, Dr Darrow completed residency in otolaryngology at the University of California, San Diego and a fellowship in pediatric otolaryngology at the Children’s Memorial Hospital in Chicago. Dr Darrow’s primary clinical and research interests are in the areas of otitis media, tonsillitis,
and vascular birthmarks. He is the founder and co-director of the EVMS Center for Hemangiomas and Vascular Birthmarks, a multidisciplinary treatment program for patients with these disorders. He has lectured locally and nationally on proper diagnosis and management of middle ear disease in children.
He has also written extensively on indications for tonsil and adenoid surgery. He has served on panels that developed guidelines for pediatric tonsillectomy, pediatric acute sinusitis, and infantile hemangiomas. Dr Darrow is the immediate past president of the Society for Ear, Nose, and Throat Adva
nces in Children. He is a former Chair of the American Academy of Pediatrics Section on Otolaryngology and has served on the executive committee of the American Society of Pediatric Otolaryngology.
兒童鼻咽部微生物及免疫病理學研究
為了解決Adenoid tonsil 的問題，作者黃建嘉 這樣論述：
背景: 腺樣體組織增生是小兒睡眠呼吸障礙與中耳積液的常見原因之一。鼻咽部微生物的無症狀定殖帶菌是許多感染疾病的先行條件。微生物定殖需要先克服宿主的免疫反應，以及所在部位微生物相的競爭。鏈球菌肺炎是鼻咽腺樣體中的主要定殖者之一，也是引起兒童中耳炎、鼻竇炎與肺炎的主要病原體之一。最近的證據顯示介白素17A (IL-17A) 介導的免疫反應與鼻咽腺樣體中肺炎球菌定殖的清除之間以及微生物相的變化存在關聯。目的: 本研究分析小兒睡眠呼吸障礙和中耳積液患童之鼻咽腺樣體組織中的介白素17A及其相關基因等免疫介子的表現，與細菌學、肺炎鏈球菌帶菌、生物膜形成情形。以及肺炎鏈球菌帶菌的影響。方法: 本研究前瞻性
的招募睡眠呼吸障礙和中耳積液的兒童。於腺樣體切除手術時，利用鼻咽拭子和取得腺樣體組織利用即時聚合酶連鎖反應、西方墨點法、免疫組織化學染色、細菌培養、掃描式電子顯微鏡、多工聚合酶連鎖反應與16S rRNA定序來分析介白素17A與相關基因的表現量、肺炎鏈球菌的帶菌與鼻咽微生物相。結果: 本研究共招募了六十六位腺樣體增生的兒童患者。我們的結果顯示， 小兒睡眠呼吸障礙患者中，增生的腺樣介白素17A, 介白素17A/介白素10, and RORγt/ Foxp3的表現量明顯高於沒有增生的檢體。鼻咽肺炎鏈球菌帶菌陽性的檢體中介白素17A和介白素17A/介白素10的表現量明顯高於陰性患者。但是，這樣的差異在
中耳積液組兒童中並不顯著。利用棉棒於鼻咽表面沾抹取得的細菌培養結果顯示兩組病人的細菌學分布沒有差異。然而腺樣體組織微生物相分析則發現在睡眠呼吸障礙這組兒童相較於中耳積液似乎有較高的生物多樣性的趨勢。(p=0.095). 鼻咽肺炎鏈球菌帶菌陽性的檢體中相較於沒有帶菌的檢體有顯著較高的生物多樣性。鼻咽肺炎鏈球菌帶菌陽性的檢體中有較多的Alloprevotella, Staphylococcus, Moraxella, 與 Neisseriaceae被偵測到。鼻咽肺炎鏈球菌帶菌陰性的檢體中則發現有較多的Dialister。結論: 這些結果顯示肺炎鏈球菌帶菌會在鼻咽腺樣體中啟動IL-17A介導的免疫反
應，可能與腺樣體的增生有關。然而，在中耳積液患者中，由於介白素17A介導的黏膜清除不充分，可能導致長期或慢性肺炎鏈球菌帶菌，以及鼻咽微生物相的改變。而肺炎鏈球菌對鼻咽微生物相的組成與生物多樣性有相當的影響力。
The Great Tonsil Massacre
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為了解決Adenoid tonsil 的問題，作者Lehr, Teresa K. 這樣論述：
The Great Tonsil Massacre uses creative non fiction to recount a public-health initiative that took place in Rochester, New York in 1920 and 1921. The narrative explores the motives of the industrialists and professionals who established two tonsil/adenoid clinics designed to benefit children whose
families were subsisting on very limited incomes. At a second level, it examines some of the reasons that may have discouraged other citizens from publicly questioning the project, even though serious risks and inflated claims were involved. Finally, it follows the tensions that arise in the O'Conne
ll family, whose members are directly affected by proponents' claims. Kate and Michael O'Connell, Timmy's mother and father, have already suffered through a botched public health event when their story opens. Memories of that event, and her and Michael's implication in it, plague Kate. Michael, a fi
rm believer in the medical miracles he has witnessed in the early 20th century, wants what is best for his son's future. At present the family can afford to sign Timmy and his younger sister Molly up for the operation, but Michael's job security is tenuous. Timmy, whose physical development lags beh
ind his peers', wants to catch up, but he is afraid of undergoing the operation. Molly adores her big brother and will follow him anywhere. Conflicts within the family and among Timmy's schoolmates escalate, causing the characters to reassess their first conclusions. An epilogue to the story examine
s the factual events as they were recorded in contemporaneous documents: meeting minutes, government reports, and newspaper articles. An appendix includes photographs of the actual clinical sites and of some of the activities that occurred in them. The narrative also focuses on the social conditions
and class-based attitudes that existed in this mid-sized manufacturing city in the early 1920s. In addition, the book addresses three questions that have persisted into the twenty-first century: 1. Can a comprehensive healthcare plan, no matter how well intentioned it may be, fit every citizen's ne
ed?; 2. Should citizens accept the pronouncements of health care professionals without question?; 3. Should parents who resist having their children immunized against communicable diseases be compelled to submit for the protection of the majority?
利用次世代基因定序找尋轉移復發頭頸癌之預測因子
為了解決Adenoid tonsil 的問題，作者呂學儒 這樣論述：
復發轉移性頭頸部鱗狀細胞癌的中位存活期僅12個月。然而，在精準醫療的年代，針對特殊臨床需求的預測指標和藥物指引卻相當少，例如是否可使用上皮生長因子受體 (EGFR) 酪氨酸激酶抑製劑 (TKI)、介入性治療遠處轉移性病灶、和及早預防與治療早期復發的病患。本自公開資料庫Sanger GDSC2 (Genomics of Drug Sensitivity in Cancer, http://www.cancerrxgene.org/)， 利用計算機運算建立一個以合成致死概念為主的藥物選擇平台，其中復發轉移頭頸癌的合成致死對並做體外驗證。我們期待推廣這個平台到癌症轉移和早期進展的藥物選擇。根據不同
的臨床需求，將檢體分為實驗組和對照組，對這些檢體給予次世代基因定序，並利用公開的資料庫，如DAVID、CPDB、和IPA，找尋其訊息傳遞路徑。共有719個合成致死對在計算機運算當中被確認，而頭頸癌的合成致死在體外驗證顯示與預測相符，故應用我們的平台可能會為對未滿足的臨床需求提供其獨特的機會。對於癌症轉移，代謝相關的訊息傳遞路徑至為重要，如branched-chain amino acid consumption，尤其是ALDH7A1突變可能是其中一個可被治療的位點；另外根據次世代基因定序的初步結果，預測轉移性病灶切除療效的基因組套也可被建立。針對早期惡化之議題，早期和晚期惡化的復發轉移頭頸癌
病患，其臨床差異已經確定，預測早期惡化的預測模型也已建立，未來將利用這些初步結果，持續找出造成早期惡化的驅動基因。我們建立並驗證了一個以合成致死為概念的藥物選擇平台。利用次世代基因定序的方式，找出不同的臨床需求的病生理特徵，並用此平台進行後續的藥物開發。而次世代定序的結果也可用來建立基因預測模型。未來仍需進行大規模驗證。