歡迎來到演算法、軟體和硬體三位一體的未來世界論文書籍站
focal neurological s的問題，透過圖書和論文來找解法和答案更準確安心。 我們找到下列懶人包和總整理
focal neurological s的問題，我們搜遍了碩博士論文和台灣出版的書籍，推薦Levin, Kerry H. (EDT)/ Chauvel, Patrick (EDT)寫的 Clinical Neurophysiology 和Ahumada, Luisa C. Busch De/ Ahumada, Jorge L.的 Contacting the Autistic Child: Five Successful Early Psychoanalytic Interventions都 可以從中找到所需的評價。
這兩本書分別來自 和所出版 。
慈濟大學 醫學科學研究所博士班 彭士奕所指導 林皓然的 應用五味子乙素於曼森血吸蟲感染之相關損傷的治療效果 （2021），提出focal neurological s關鍵因素是什麼，來自於血吸蟲病、五味子乙素、吡喹酮、曼氏血吸蟲、纖維化。
而第二篇論文國立陽明交通大學 分子醫學博士學位學程 陳儀莊所指導 李晏嬅的 PIAS1 基因變異體在亨丁頓舞蹈症之功能性鑑定 （2021），提出因為有 亨廷頓舞蹈症、醯胺麩胺酸疾病、基因調飾因子、PIAS1基因變異體、小 泛素的重點而找出了 focal neurological s的解答。
Clinical Neurophysiology
為了解決focal neurological s 的問題，作者Levin, Kerry H. (EDT)/ Chauvel, Patrick (EDT) 這樣論述：
Clinical Neurophysiology: Diseases and Disorders, the latest release in the Handbook of Clinical Neurology series, reviews the current practice of clinical neurophysiology in the laboratory, by the bedside, and in the operating room or intensive care unit. The volume is organized into sections focus
ed on diseases of the central and peripheral nervous systems, sleep disorders, and autonomic disorders. Among the CNS topics covered are epilepsy, altered states of consciousness, disorders of cognition, brain death, demyelinating diseases, stroke, pain, movement disorders, vestibular disease, and a
uditory disorders. Peripheral nervous system topics include focal mononeuropathies, generalized polyneuropathies, muscle diseases, hyperexcitability states, neuromuscular junction disorders, anterior horn cells diseases, and cranial neuropathies. There are also chapters on sleep apneas, hypersomnias
, parasomnias, and circadian rhythm disorders. Autonomic topics include primary autonomic failure, multisystem atrophy, and postural orthostatic tachycardia syndrome. Provides an up-to-date review on the practice of the neurophysiological techniques used in the assessment of neurological diseasesExp
lores the electrophysiological techniques used to better understand neurological function and dysfunction of the central and peripheral nervous systemsDiscusses monitoring neurologic function in the intensive care unit and the assessment of suspected brain deathIncludes discussions of various newer
techniques, including functional brain mapping, stereo EEG, motor evoked potentials, magnetoencephalography, laser evoked potentials, and transcranial magnetic stimulation
應用五味子乙素於曼森血吸蟲感染之相關損傷的治療效果
為了解決focal neurological s 的問題，作者林皓然 這樣論述：
血吸蟲病（Schistosomiasis）是世界上僅次於瘧疾的最重要的寄生蟲病。臨床上，血吸蟲病患會出現肝臟或其他器官纖維化的症狀。目前，血吸蟲病患者以吡喹酮（Praziquantel；PZQ）治療為主。然而，雖然 PZQ 能有效殺死血吸蟲蟲，它不能防止病患的再次感染或治療肝臟纖維化。而目前的治療方法也不足以治癒肝臟纖維化。除此之外，目前已在眾多體內和體外的研究中發現了血吸蟲對PZQ的抗藥性。因此，我們迫切的需要尋找新的有效治療藥物。目前五味子植物中的五味子乙素（Schisandrin B；Sch B）已被證明可以預防各種不同的肝臟損傷。因此，我們在此研究使用Sch B治療因曼森血吸蟲（Sc
histosoma mansoni）誘發的各種器官損傷的潛力。本研究的結果顯示，Sch B可通過抑制發炎小體的活化和細胞凋亡，以及調節免疫反應，來治療因曼森血吸蟲誘導的肝臟纖維化。此外，Sch B可破壞雄性成蟲，從而有助於減少產卵量並減輕病變。我們進一步的實驗發現，PZQ-Sch B 治療可對血吸蟲病產生更有效的治療反應。這種治療策略可以保護與曼森血吸蟲感染相關的器官損傷，包括肝臟、脾臟、腸道和肺部。此外，PZQ-Sch B的治療提高了感染小鼠的存活率，並且達到更好的預後。總的來說，Sch B對於治療血吸蟲病相關的肝臟損傷和全身併發症可能是一個很有效的藥物。
Contacting the Autistic Child: Five Successful Early Psychoanalytic Interventions
為了解決focal neurological s 的問題，作者Ahumada, Luisa C. Busch De/ Ahumada, Jorge L. 這樣論述：
Amid long-standing controversy on their causes, which most regard as neurological, and despite their increasing social impact, there has been scant progress in the therapy of the autistic spectrum disorders. Currently fashionable attempts at treatment through behavioural-cognitive focal approache
s do not seek resolution, only re-education and rehabilitation. Contacting the Autistic Child explores the clinical process in the early psychoanalytic treatment of autistic children. Organised around five detailed clinical case studies, and drawing on the ideas of major clinicians in child analysis
such as Tustin, Winnicott and Alvarez, this book sets out a clear programme for working with and understanding autistic children in a psychoanalytic setting, with a particular focus on issues of clinical technique but also conceptual matters. Working on the notion that autistic disorders come to be
- as Winnicott and Tustin saw it - from an early rupture of the affective communication between baby and mother, this book aims at reinstating such communication in the child-analyst interaction. By way of detailed description of what goes on in the analytic link, the authors strive to make the rea
der share in what goes on in the clinical setting, evincing how, though at times excruciatingly hard on the therapist, resolution is attainable. Once the "primal dialogue" - to use Ren Spitz's terms - is reinstated in a stable way in session, it flows by itself into the family ambience. The clinica
l accounts of this book make the argument that psychoanalysis, carried along Tustin's technical lines, and subject to the proviso that treatment starts early, preferably in the first three years of life, is the treatment of choice for autistic spectrum disorders. The strong methodological narrative
is important and notable in light of the doubts, criticism and uncertainty that have surrounded the psychoanalytic treatment of autism. This novel, highly detailed narrative of five successful early treatments aims to help dispel the pessimism pervading the field and help to redress the lives of ma
ny more children. Contacting the Autistic Child will appeal to psychoanalysts and psychoanalytic psychotherapists endeavouring to obtain results in a major area lacking resolutive approaches.
PIAS1 基因變異體在亨丁頓舞蹈症之功能性鑑定
為了解決focal neurological s 的問題，作者李晏嬅 這樣論述：
亨廷頓舞蹈症 (HD) 是一種遺傳的神經退化性疾病。此症是由HTT基因中的CAG三核苷酸重複 (CAG repeat) 擴增引起的神經退化性疾病。 CAG三核苷酸重覆數的變異相關的醯胺麩胺酸 (polyQ) 疾病，包括 HD 和脊髓小腦共濟失調 3 型 (SCA3) ，CAG三核苷酸重覆數的長度是決定發病年齡 (AO) 的主要因素。實際上，一些患者的發病年齡可能偏離平均發病年齡 (average AO)， 顯示基因調飾因子的存在，並可以調控疾病的進展、嚴重度和發病年齡。為了鑑識醯胺麩胺酸疾病中常見的基因調飾因子，我們團隊採集了 337 名 HD 或 SCA3 患者的血液檢體，並檢測與蛋白質穩
定相關的 583 個基因的標靶定序與生物資訊分析。從較晚發病的患者中, 我們團隊一共找到16個基因被鑑定可調控發病年齡的基因調飾因子。我們進一步研究了PIAS1的基因變異體 (gene variant)，因為PIAS1是亨廷頓蛋白(HTT)的 E3小泛素連接酶 (SUMO ligase)。生化分析的結果顯示，相對於 PIAS1WT，PIAS1S510G 與突變亨廷頓蛋白(mHTT) 的交互作用的能力降低，導致突變亨廷頓蛋白的小泛素化(SUMOylation)水平降低，進而減少突變亨廷頓蛋白的累積。透過基因編輯技術，我們把HD小鼠模型 (R6/2)的Pias1WT修改為Pias1S510G，發現
小鼠的疾病症狀也有所改善。我們的研究結果證明，PIAS1可以調控醯胺麩胺酸疾病的發病年齡，和疾病小鼠的疾病嚴重度。研究蛋白質平衡網絡中的基因調飾與其功能，可以提供創新的疾病治療方案。